MLADA OGULINSKA ZNANSTVENICA U "RUĐERU BOŠKOVIĆU"
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Naša Ogulinka kreće u bitku s jednim od danas u medicimi navjećih problema.
Mlada znanstvenica dr. sc. Nikolina Udiković Kolić, kćerka pok. vlasnika elektroobrta Udiković, koja radi na Institutu "Ruđer Bošković", dobila je donaciju vrijednu 120 tisuća kuna, za projekt "Proučavanje povezanosti zagađenja antibioticima i razvoja rezistencije na antibiotike u ljudskih patogena". Donaciju je dobila u sklopu programa Zaklade Adris kojom se podupire inovativnost i kvaliteta u radu uspješnih znanstvenika, učenika, studenata i ostalih inovatora.
Otpornost bakterija na antibiotike trenutno je jedan od najvećih medicinskih problema s kojim se suočava svijet, a zbog toga je ugroženo liječenje brojnih, dosad izlječivih zaraznih bolesti. Bakterije su s vremenom razvile mehanizme otpornosti na sve grupe antibiotika koji se upotrebljavaju pa zbog toga oni postaju nedjelotvorni. Borba protiv antibiotičke rezistencije ubraja se u prioritete Svjetske zdravstvene organizacije. Projektom kojim se bavi mlada znanstvenica Nikolina Udiković - Kolić, istraživat će se mehanizmi rezistencije na antibiotike.
Dobiveni će podaci poduprijeti napore za razvoj strategija za suzbijanje širenja antibiotičke rezistencije putem okoliša. Pokazalo se je da okoliš može služiti kao rezervoar rezistentnih mikroorganizama koji mogu uzrokovati bolest kod ljudi, pa je pored bolničkih sredina, rezistenciju neophodno pratiti i u okolišu.
"Od posebne je važnosti vodeni okoliš koji je pod utjecajem zagađenja antibioticima putem otpadnih voda iz farmaceutske industrije, jer takve otpadne vode mogu sadržavati smjese antibiotika u visokim koncentracijama. Zato okoliš izložen ispustu ovakvog otpada predstavlje "vruće točke" za umnožavanje postojećih i razvoj novih gena za antibiotičku rezistenciju, te time predstavlja potencijalnu opasnost za zdravlje ljudi", objasnila je znanstvenica Udiković - Kolić, iz Zavoda za istraživanje mora i okoliša Instituta Ruđer Bošković.
9. 11. 2017,
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'all-out' sprints on a Wingate bicycle ergometer, on which the brake band of
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of 15-20 min between consecutive sprints. Thigh muscle biopsies were
taken before and after the training period and were analysed for fibre
types using a myofibrillar ATPase stain. The
proportion of type I fibres decreased from 57 to 48% (P less than 0.05) and type
IIA fibres increased from 32 to 38% (P less than 0.05).
This study indicates that it is possible to achieve a fibre type transformation with high-intensity training.
The effect of two-legged 'sprint' training
on muscle fibre type composition may be related to a
changed pattern of muscle fibre activation (e.g.
an increased stimulation frequency). A change in fibre activation frequency may induce an increased
synthesis of type II fibre myosin (fast myosin).
Hormonal influences such as enhanced adrenergic stimulation of the muscle fibres cannot be excluded as
a contributing factor, however.
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